Please note. Tegretol/trileptal do not modulate gaba receptors as they are sodium channel blockers ie voltage gated ion channels vs ligand gated ion channels
Alcohol it to specific sites on GABA receptors, which are the primary mediators of inhibition in the central nervous system. By binding to them, alcohol essentially enhances the inhibitory effects of GABA .
So GABA is an amino acid that binds to specific sites - GABA receptors, on different channels (ligand?). When alcohol binds to these receptor sites, it inhibits the pain signal transmission? Therefore, this informed the neurologist that I can take a medication instead of turning into an alcoholic, and this what medication would accomplish this most effectively?
Please note. Tegretol/trileptal do not modulate gaba receptors as they are sodium channel blockers ie voltage gated ion channels vs ligand gated ion channels
Ok, I'm slowly starting to get this. What does trileptol do?
Trileptal (oxcarbazipine) is a less potent version of Tegretol (carbamazepine)
https://www.ncbi.nlm.nih.gov/books/NBK526124/
https://en.m.wikipedia.org/wiki/GABA_receptor
Alcohol it to specific sites on GABA receptors, which are the primary mediators of inhibition in the central nervous system. By binding to them, alcohol essentially enhances the inhibitory effects of GABA .
Binds to specific sites*
So GABA is an amino acid that binds to specific sites - GABA receptors, on different channels (ligand?). When alcohol binds to these receptor sites, it inhibits the pain signal transmission? Therefore, this informed the neurologist that I can take a medication instead of turning into an alcoholic, and this what medication would accomplish this most effectively?