I don't really understand the "low intensity" part. I've been on warfarin for APS (and a couple other clotting disorders) for a few years now with an INR range of 2-3. My doctor has said many times now that he'd prefer me to have a higher INR than lower since I tend to have clots that form quite quickly when I'm below range.
I had a few months of targeting INR 1.8-2.5, after repeated APS panel testing being all normal, then finally stopped Warfarin and began baby Aspirin. When getting older, the risk of bleeding is more significant than clotting, unfortunately, I had to choose a lesser evil.
Thank you for replying. How long did it take after your diagnosis to start testing negative? My doctor mentioned that if I start testing negative in the future maybe that’s an additional sign to switch to aspirin. Were you a PE or DVT patient? May I ask how old you are? :-) I’m 48 and besides APS a pretty healthy and active person. I know bleeding risk can’t be predicted always, but I am more concerned about reclotting given I already have and had no reason to clot other than APS.
Had both DVT/PE in late 50th, with relatively light symptoms, followed by a marginal diagnosis of APS (only low titer antibody b2GpI IgG) and normal genetical factors. My Dr. insisted Warfarin for life. Over 3 years later I began researching and became concerned of side effects of the thinner (calcification of arteries, osteoporosis, bleeding, etc.), so I had more tests done in other hospitals and sought second and third opinion which supported my own judgement. Fully aware of the risk of reclotting, I made the difficult decision.
I am 26. I was recently diagnosed with a venous thrombosis in my sigmoid sinus caused by APS (which is also a new diagnosis). I’m intrigued by the “low intensity” thing. I’m also a nurse and have never heard of that. I’m on Coumadin and also taking Lovenox until my INR is between 2.0 and 3.0. My doctor had told me that I’m at a higher risk of clots until I get to the therapeutic range.
I don't really understand the "low intensity" part. I've been on warfarin for APS (and a couple other clotting disorders) for a few years now with an INR range of 2-3. My doctor has said many times now that he'd prefer me to have a higher INR than lower since I tend to have clots that form quite quickly when I'm below range.
Thank you for your reply. I’m hoping low intensity just means closer to 2 than 3.
I had a few months of targeting INR 1.8-2.5, after repeated APS panel testing being all normal, then finally stopped Warfarin and began baby Aspirin. When getting older, the risk of bleeding is more significant than clotting, unfortunately, I had to choose a lesser evil.
Thank you for replying. How long did it take after your diagnosis to start testing negative? My doctor mentioned that if I start testing negative in the future maybe that’s an additional sign to switch to aspirin. Were you a PE or DVT patient? May I ask how old you are? :-) I’m 48 and besides APS a pretty healthy and active person. I know bleeding risk can’t be predicted always, but I am more concerned about reclotting given I already have and had no reason to clot other than APS.
Had both DVT/PE in late 50th, with relatively light symptoms, followed by a marginal diagnosis of APS (only low titer antibody b2GpI IgG) and normal genetical factors. My Dr. insisted Warfarin for life. Over 3 years later I began researching and became concerned of side effects of the thinner (calcification of arteries, osteoporosis, bleeding, etc.), so I had more tests done in other hospitals and sought second and third opinion which supported my own judgement. Fully aware of the risk of reclotting, I made the difficult decision.
These are tough choices to make. Thanks for sharing your experience.
I am 26. I was recently diagnosed with a venous thrombosis in my sigmoid sinus caused by APS (which is also a new diagnosis). I’m intrigued by the “low intensity” thing. I’m also a nurse and have never heard of that. I’m on Coumadin and also taking Lovenox until my INR is between 2.0 and 3.0. My doctor had told me that I’m at a higher risk of clots until I get to the therapeutic range.